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The redox regulation of PI 3-kinase-dependent signaling
Nick R Leslie
School of Engineering & Physical Sciences
Institute of Biological Chemistry, Biophysics and Bioengineering
Research output
:
Contribution to journal
›
Article
›
peer-review
124
Citations (Scopus)
Overview
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Dive into the research topics of 'The redox regulation of PI 3-kinase-dependent signaling'. Together they form a unique fingerprint.
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Medicine and Dentistry
Phosphoinositide 3-Kinase
100%
Signal Transduction
66%
Kinase Signaling
50%
Regulatory Mechanism
50%
Oxidative Stress
33%
Protein
16%
Lipid
16%
Cell Growth
16%
Stimulation
16%
Cell Motility
16%
Growth Factor
16%
Cell Survival
16%
Cytokine
16%
Phosphatidylinositol 3,4,5 Trisphosphate 3 Phosphatase
16%
Protein Tyrosine Phosphatase
16%
Redox Signalling
16%
Insulin Resistance
16%
Muscle
16%
Transduction Mechanism
16%
Neuroscience
Phosphoinositide 3-Kinase
100%
Cell Signaling
66%
Oxidative Stress
33%
Growth Factor
33%
Lipid
16%
Reactive Oxygen Species
16%
Cell Growth
16%
Cell Motility
16%
Phosphatidylinositol 3,4,5 Trisphosphate 3 Phosphatase
16%
Cell Survival
16%
Protein Tyrosine Phosphatase
16%
Protein
16%
Pharmacology, Toxicology and Pharmaceutical Science
Phosphatidylinositol 3 Kinase
100%
Reactive Oxygen Metabolite
33%
Protein
16%
Cytokine
16%
Lipid
16%
Phosphatidylinositol 3,4,5 Trisphosphate 3 Phosphatase
16%
Protein Tyrosine Phosphatase
16%
Cellular Processes
16%
Insulin Resistance
16%
Reactive Nitrogen Species
16%
Biochemistry, Genetics and Molecular Biology
Signal Transduction
66%
Regulatory Mechanism
50%
Oxidative Stress
33%
Cell Survival
16%
Cytokine
16%
Cell Growth
16%
Cell Motility
16%
Insulin Resistance
16%
Species
16%
Nitrosylation
16%